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KMID : 0358520090210030289
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Korean Journal of Occupational and Environmental Medicine
2009 Volume.21 No. 3 p.289 ~ p.292
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A Case of Cerebellar Dysfunction After Acute Organotin Poisoning
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Kim Suk-Hwan
Kim Yang-Ho
Yoo Cheol-In
Kwon Jee-Hyun
Bae Jin-Ho
Weon Young-Cheol
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Abstract
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Background: We performed a follow-up study of an acute organotin poisoning case on the neurological sequelae that remained after 3 years since diagnosis.
Case: In the previous study, a 43-year-old male was reported for acute organotin poisoning. After 3 years, the neurological sequelae were studied using neurological examination, cognitive tests, neurobehavioral tests (by CAYSYS 2000TM), brain magnetic resonance imaging (MRI), and 18F-fluorodeoxyglucose (18F-FDG) PET/CT. Upon neurological examination and other tests, orientation was improved compared to that 3 years before, but cerebellar ataxia, dysmetria and dysdiadochokinesia remained the same, with little change. Brain MRI showed cerebellar atrophy. 18F-FDG PET/CT revealed mildly decreased metabolic activity in the pons and in both cerebellar hemispheres.
Discussion: This is the first brain MRI study on cerebellar atrophy caused by organotin poisoning. Cerebellar ataxia was confirmed by decreased metabolic activity in 18F-FDG PET/CT.
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KEYWORD
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Organotin, Cerebellar ataxia, Fluorodeoxyglucose F18
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